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. 2019 Nov 29;10:362. doi: 10.1186/s13287-019-1490-8

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — fMSCs decreased oxidative damage, increased oxidative protection, improved anti-apoptosis rates, and inhibited apoptosis in the POI mouse model. a fMSCs were injected into the POI mouse model by tail intravenous. b FACS analysis of ROS expression at 2 weeks in different treatment groups. c–h ELISA analysis of MDA, SOD, CAT, LDH, GR, and GPx levels at 8 weeks in different treatment groups. i qPCR detection of SURVIVIN and BCL2 levels in different treatment groups. j qPCR analysis of CASPASE-3 and CASPASE-9 levels in different treatment groups. k Western blot analysis of SURVIVIN, BCL2, CASPASE-3, and CASPASE-9 in different treatment groups. Data are represented as the mean ± SD. ***p < 0.001 (compared with the NG group)