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. 2019 Nov 29;16:149. doi: 10.1186/s12985-019-1258-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Replication of wild-type and mutant H1N1 influenza A viruses in the absence (a) and presence of 5 nM oseltamivir carboxylate (b) in Calu-3 cells. The results are expressed as log₁₀PFU/ml from three to four independent experiments performed on different days. *P < 0.05, compared to the values for the wild-type CA/04 virus (one-way ANOVA)