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. 2019 Nov 5;18(6):6483–6490. doi: 10.3892/ol.2019.11060

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — MIR100HG expression was significantly upregulated in CRC tissues and could predict a poor prognosis. (A) Expression levels of MIR100H in 40 paired CRC and adjacent noncancerous tissues. (B) MIR100HG expression level in low clinical stage (I–II) CRC tissues and advanced stage (III–IV) CRC tissues was analyzed. Kaplan-Meier analysis and log-rank test of the (C) overall survival and (D) disease-free survival in patients with CRC according to MIR100HG expression level. AJCC, American Joint Committee on Cancer; CRC, colorectal cancer; MIR100HG, mir-100-let-7a-2-mir-125b-1 cluster host gene. **P<0.01 vs. normal tissue.