Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2019 Oct 29;60(12):2090–2101. doi: 10.1194/jlr.P119000226

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2019 Kang and Sung.

Published under exclusive license by The American Society for Biochemistry and Molecular Biology, Inc.

PMC Copyright notice

Fig. 3. — Contributions of marginal associations and gene-obesity interactions to the risk of dyslipidemia. The pie plots describe the proportion of phenotypic variation attributable to the overall genetic variation (total heritability), genetic markers assayed by SNP arrays (SNP-based heritability), and the combined set of both GWAS-identified and novel G×E variants. The bar plots, on the other hand, show the proportion of genetic variation explained by marginal and gene-obesity interactive effects. Parts (A) to (C) describe the genetic contributions to abnormal TG due to the interplay between genes and obesity indices classified by BMI (A), WC (B), and WHR (C). D: The genetic contributions to abnormal Remnant-C due to the interplay between genes and obesity indices classified by WHR; further details are presented in Table 3.