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. 2019 Nov 1;158:107728. doi: 10.1016/j.neuropharm.2019.107728

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© 2019 The Authors

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

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Fig. 3 — Levels of receptor proteins in temporal lobe that showed significant changes between AMC and RISE rats at different stages of epileptogenesis.

Top panels: Representative Western Blots of hippocampal samples blotted for AMPAR subunits (GluA1, GluA2 and GluA3), KAR subunit GluK5, NMDAR subunits (GluN1, GluN2A, GluN2B), Group 1 metabotropic glutamate receptor subtypes (mGluR1α and mGluR5), and GABA_AR subunit β3 (GABA_A β3). β-actin and α-GAPDH were used as loading controls.

Bottom panels: Quantification of immunoreactive bands normalised to their respective loading controls. Error bars represent the mean ± SD from 4 to 5 samples, with the mean of the control group set to 1. *P < 0.05, **P < 0.01, ***P < 0.001 vs Ctrl (Unpaired t-test).