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. 2019 Dec 5;10(12):930. doi: 10.1038/s41419-019-2171-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — a Relative expression level of TET1 in HEEpiC and four esophageal cancer cell lines. b Relative expression level of TET1 in ESCC tissues and corresponding normal tissues. c Relative expression level of UTX in HEEpiC and four esophageal cancer cell lines. d Relative expression level of UTX in ESCC tissues and corresponding normal tissues. e The influence of TET1, UTX, and ZNF667-AS1 on Eca109 cells viability. *P < 0.05, pcDNA3.1-TET1 or pcDNA3.1-UTX compared with pcDNA3.1-NC; pcDNA3.1-TET1 + pcDNA3.1-UTX compared with pcDNA3.1-TET1 or pcDNA3.1-UTX; pcDNA3.1-TET1 + pcDNA3.1-UTX + pcDNA3.1-ZNF667-AS1 compared with pcDNA3.1-TET1 or pcDNA3.1-UTX or pcDNA3.1-TET1 + pcDNA3.1-UTX; pcDNA3.1-TET1 + pcDNA3.1-UTX + shRNA-ZNF667-AS1-1 compared with pcDNA3.1-TET1 + pcDNA3.1-UTX + pcDNA3.1-ZNF667-AS1. f The influence of TET1, UTX, and ZNF667-AS1 on Eca109 cells invasive ability. The experiments are representative of three independent experiments with similar results. *P < 0.05.