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. 2019 Nov 21;21(1):320–328. doi: 10.3892/mmr.2019.10839

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Copyright: © Lu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Effect of CMR on Nissl bodies in DRG neurons in diabetic rats. DRG neurons were separated and extracted from the spinal cord and fixed with 4% paraformaldehyde for frozen sectioning. Morphological changes of DRG neurons were observed, including Nissl bodies and the size of neurons. White asterisks indicate Nissl bodies. Scale bar, 50 µm. ***P<0.001 vs. control; ^##P<0.01 vs. model; ^ΔP<0.05 vs. CMR. CMR, Cortex Mori Radicis extract; TRPC1, transient receptor potential canonical channel 1; si-, small interfering RNA; DRG, dorsal root ganglia.