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. 2019 Dec 6;9:18525. doi: 10.1038/s41598-019-53682-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(a) Left row: Raw simulated MEG signals for high (50%) and low (10%) percentage of chandelier cells among total inhibitory cells for a drive frequency of 40 Hz and the two network conditions (‘control’: black traces; ‘schizophrenia-like’: red traces). Right row: Power spectral density of the MEG signals of the left row. (b) Comparison of the 5 entrainment measures between control (black) and ‘schizophrenia-like’ (red) for high and low ChC percentages. Note that ‘schizophrenia-like’ here means that IPSC decay times at chandelier cell synapses were increased from 8 ms to 28 ms. All traces here depict the mean signal averaged over 20 trials with different realisations of the Poisson process underlying the generation of noise. Importantly, averaging was performed in the time domain prior to a transformation into the frequency domain via Fourier transform, which is also the standard procedure in experiments.