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. 2019 Dec 9;10(12):936. doi: 10.1038/s41419-019-2177-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Immunoblot analysis clarified that LCN2 was successfully inhibited or overexpressed in OSCC cells; b Gradient concentration of the cisplatin-treated OSCC shLCN2 cell line (MTS assays were performed to determine the enhancement of cisplatin sensitivity); c Gradient concentration of the cisplatin-treated OSCC LCN2-ov cell line (MTS assays were performed to determine the inhibition of cisplatin sensitivity); d Flow cytometry detected the cisplatin effect on the OSCC shLCN2 cell line; e Flow cytometry detected the cisplatin effect on the OSCC LCN2-ov cell line; f Xenograft growth statistics from groups injected with the CAL-27 shLCN2 cell line; g Xenograft growth statistics from groups injected with the CAL-27 LCN2-ov cell line.