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. 2019 Dec 9;9:18628. doi: 10.1038/s41598-019-55203-w

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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mam genetically interacts with aos during the OR process. (a–h) Adult eye sections with ommatidial orientation schematics (arrows as in Fig. 1) and orientation angle histograms of eyes of the genotypes indicated. All genotypes are mδ0.5-Gal4 > mam^RNAi (BL28046) in the following genetic backgrounds: (a,b) +/+ (wild type); (c,d) aos^Δ7/+, (e,f) Df(3 L)BSC562/+ (deletion of aos gene), and (g,h) nmo^DB/+. Asterisks denote significance by chi-square test (***p < 0.0005). Note robust enhancement of the mδ0.5-Gal4 > mam^RNAi rotation phenotype by both aos^−/+ genotypes. See supplemental material for additional genotypes.