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. 2019 Dec 9;17(4):1559325819894148. doi: 10.1177/1559325819894148

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© The Author(s) 2019

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 5. — Concentration–response curves induced by cumulative addition of dexmedetomidine (A), followed by N^ω-nitro-l-arginine methyl ester (l-NAME, 10⁻⁴ M) (B) in endothelium-intact rat aortae with (N = 15) or without (n = 13) linolenic acid and endothelium-denuded rat aortae (N = 15). l-NAME (B) was added only to endothelium-intact rat aortae with or without linolenic acid after dexmedetomidine (10⁻⁶ M) produced maximal contraction. Data are shown as mean ± standard deviation and expressed as the percentage of maximal contraction induced by isotonic 60 mM KCl. N indicates the number of isolated rat aortae. A, *P < .01 and ^† P < .001 versus control. B, *P < .001 versus dexmedetomidine (10⁻⁶ M) alone.