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. 2019 Sep 13;33(11):12853–12872. doi: 10.1096/fj.201900057R

Figure 8.

Figure 8

Mitochondrial oxidative capacity parallels TRPC1 channel expression and magnetic sensitivity. A–C) Mitochondrial OCR in response to PEMF exposure in wild-type C2C12 myoblasts (A) and in CRISPR/Cas9 TRPC1-silenced clones c60 (B) and c61 (C) (also see Table 2). D–F) ECAR in wild-type C2C12 myoblasts (D) and in CRISPR/Cas9 TRPC1-silenced clones c60 (E) and c61 (F). G–I) Mitochondrial OCR from wild-type C2C12 myoblasts in response to 10 min exposures to 0 (G), 1.5 (H), or 3 mT (I) amplitude PEMFs in the absence (solid symbols) or presence (hatched symbols) of SKF-96365 (SKF; 50 µM) as indicated. J–L) ECAR in wild-type C2C12 myoblasts in response to 10 min exposures to 0 (J), 1.5 (K), or 3 mT (L) amplitude PEMFs in the absence (solid symbols) or presence (hatched symbols) of SKF (50 µM) as indicated.