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. 2007 Oct 1;22(10):1659–1673. doi: 10.1007/s00467-007-0482-6

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© IPNA 2007

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Fig. 1 — Schematic representation of molecular (or transport) mechanisms in the proximal tubule related to Dent’s disease and hereditary hypophosphatemic rickets with hypercalciuria. • low molecular weight proteins; NHE-3 Na⁺-H⁺ exchanger; I, IIa, IIc, and III Na/Pi type I, type IIa, type IIc and type III cotransporters; NBC-1 Na⁺-HCO⁻ ₃ cotransporter; CLC-5 chloride channel-5; AA amino acids; X ⁻ anion; Pi phosphate; CA II cytoplasmic carbonic anhydrase; CA IV membrane-bound carbonic anhydrase

Inline graphic — Schematic representation of molecular (or transport) mechanisms in the proximal tubule related to Dent’s disease and hereditary hypophosphatemic rickets with hypercalciuria. • low molecular weight proteins; NHE-3 Na⁺-H⁺ exchanger; I, IIa, IIc, and III Na/Pi type I, type IIa, type IIc and type III cotransporters; NBC-1 Na⁺-HCO⁻ ₃ cotransporter; CLC-5 chloride channel-5; AA amino acids; X ⁻ anion; Pi phosphate; CA II cytoplasmic carbonic anhydrase; CA IV membrane-bound carbonic anhydrase