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. 2019 Dec 10;9:18722. doi: 10.1038/s41598-019-54286-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Whole exome mutational burden of intramedullary spinal cord tumors. (A) astrocytic tumors, (B) ependymomas. One subependymoma was discovered to harbor 315 unique somatic mutations and was held out from this analysis. Unpaired t-test was used to test for significance between high-grade gliomas (HGG) and low-grade gliomas (LGG). ANOVA was used to test for significance between ependymoma subtypes. SNVs, single-nucleotide variants. GBM, glioblastoma. AA, anaplastic astrocytoma. LG2, Grade II glioma. PA, pilocytic astrocytoma. Classic, classic ependymoma. MPE, myxopapillary ependymoma. SE, subependymoma.