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. Author manuscript; available in PMC: 2019 Dec 11.
Published in final edited form as: Clin Gastroenterol Hepatol. 2019 Feb 8;17(12):2541–2551.e2. doi: 10.1016/j.cgh.2019.02.005

Table 4.

Association Between ALT Flares >10× ULN and Clinical Outcomes for HBeAg-Negative Participants

Clinical outcome Participants free of ALT flares Participants with ALT flares P value
Treatment initiation N = 1172 N = 37 <.0001
N PYR Rate N PYR Rate
110 1190.7 9 17 24.7 69
Specific treatments
 ETV or TDF 87 11
 Other NA 8 5
 PEG-IFN 10 0
 Combination 5 1
HBV DNA decline (>1 log) N = 1115 N = 33
N PYR Rate N PYR Rate .003
Untreateda 203 889.7 23 10 16.9 59
Treateda 64 48.5 132 14 10.8 129 .94
HBsAg loss N = 904 N = 31
N PYR Rate N PYR Rate
Untreateda 15 761.8 2 1 11.0 9 .14
Treateda 0 41.4 0.0 0 9.3 0.0 1.0
Disease progressionb 2 0 1.0

ALT, alanine aminotransferase; Combination, being in >1 of the previous therapy groups in the course of the 48-week window; HBeAg, hepatitis B e antigen; HBsAg, hepatitis B surface antigen; HBV, hepatitis B virus; IFN, interferon (including pegylated [PEG] form); NA, nucleoside analogue (entecavir and tenofovir); Other NA, telbivudine, adefovir, emtricitabine, truvada, lamivudine; PYR, person-years; ULN, upper limit of normal.

a

Event rates per 100 PYR. Event must occur within 48 weeks (± 12-week window) of ALT flare. Participants may contribute PYR as both treated and untreated.

b

Disease progression is defined as composite outcome of hepatic decompensation (variceal bleeding, ascites, hepatic encephalopathy), death, or transplant.