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. 2019 Dec 11;25:54. doi: 10.1186/s10020-019-0122-1

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Oxidative stress and apoptosis were observed in intestinal tissue of burn model rat. a: The total sulfhydryl (TSH) expression was detected by spectrophotometry. The malondialdehyde (MDA) expression was detected by spectrophotometry. b: The malondialdehyde (MDA) expression was detected by spectrophotometry. c: The superoxide dismutase (SOD) expression was detected by spectrophotometry. d: The catalase (CAT) expression was detected by spectrophotometry. e The apoptosis was detected by flow cytometry. f: ED inhibited the apoptosis caused by burn injury. g: The Bcl-2, cytochrome c, C caspase-9 and C caspase-3 expressions were detected by Western blotting. h: ED decreased cytochrome c, C caspase-9 and C caspase-3 expressions and increased the Bcl-2 expression. ^*P < 0.05 vs. Sham, ^**P < 0.01 vs. Sham, ^##P < 0.01 vs. Model