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. 2019 Oct 30;43(1):188–200. doi: 10.3892/or.2019.7394

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Gene expression analysis of IDH1^Wt and IDH1^Mut glioma cells under hypoxia. A previously published database (GSE118683) reporting the profiling of glioma cells grown in hypoxic vs. normoxic conditions in vitro was used. (A) Heatmap of differentially expressed genes in IDH1^Wt and IDH1^Mut glioma cells in hypoxic conditions. (B) GO term classification of differentially expressed genes. Count represents the number of genes annotated by gene ontology database to each of the GO terms. (C) KEGG pathway analysis of differentially expressed genes. IDH1, IDH1, isocitrate dehydrogenase 1; Wt, wild-type; Mut, mutant; GO, gene ontology; KEGG; Kyoto Encyclopedia of Genes and Genomes; BP, biological process; MF, molecular function; CC, cellular component.

Figure 3. — Gene expression analysis of IDH1^Wt and IDH1^Mut glioma cells under hypoxia. A previously published database (GSE118683) reporting the profiling of glioma cells grown in hypoxic vs. normoxic conditions in vitro was used. (A) Heatmap of differentially expressed genes in IDH1^Wt and IDH1^Mut glioma cells in hypoxic conditions. (B) GO term classification of differentially expressed genes. Count represents the number of genes annotated by gene ontology database to each of the GO terms. (C) KEGG pathway analysis of differentially expressed genes. IDH1, IDH1, isocitrate dehydrogenase 1; Wt, wild-type; Mut, mutant; GO, gene ontology; KEGG; Kyoto Encyclopedia of Genes and Genomes; BP, biological process; MF, molecular function; CC, cellular component.