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. 2019 Dec 13;10(12):951. doi: 10.1038/s41419-019-2185-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Left: representative images of the immunofluorescence analysis of HP1 levels (green) in MRC5 and NBS cells. Bar: 20 μm. Right: Distribution of HP1α corrected nuclear fluorescence intensity in MRC5 and NBS1 cells. The horizontal lanes indicate the mean values derived from the analysis of 100 cells/experimental point in three independent experiments ±S.D. (t Student’s Test; ***p < 0.001). b WB analysis of HP1α levels in fibroblast and lymphoblastoid cells (LCLs). The levels of HP1α and NBS1 in NBS cells were compared to their corresponding controls (i.e., MRC5 for NBS1 fibroblasts; Ctrl for NBS1–1 and NBS1–2 lymphoblastoid cells); β-actin was used as the loading control.