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. 2019 Nov 26;8(23):e013112. doi: 10.1161/JAHA.119.013112

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© 2019 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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MicroRNA‐101 (miR‐101) could target runt‐related transcription factor 1 (RUNX1), which was an upregulated gene in myocardial infarction (MI). A, The heatmap of the top 15 differentially expressed genes (DEGs) in the microarray data GSE46395. The abscissa refers to the sample number, the ordinate refers to the DEGs, and the upper right histogram represents color grade. Each rectangle in the image represents the expression of a sample. B, Gene interaction network of the DEGs and MI genes, in which the diamonds refer to MI genes and the circles refer to DEGs. C, Expression of 8 nonoverlapping miRs in myocardial tissues in the sham‐operated and MI rats determined by reverse transcription quantitative polymerase chain reaction. BCR indicates B cell receptor; DBN1, Drebrin; FLNC, filamin C; LTB, heat‐lable enterotoxin B unit; RTN4, reticulon 4; SH2D5, SH2 domain‐containing 5; SLFN2, Schlafen; TTC9, Tetratricopeptide repeat domain 9; Tubb6, beta 6 class V β‐tubulin.