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. 2019 Dec 16;11:27. doi: 10.1186/s11689-019-9290-0

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Increase in activity of the Gfap promoter during early postnatal development of a mouse model of Alexander disease. Promoter activity (monitored through expression of a Gfap-luciferase reporter) in mutants rises above that in wild-type mice between postnatal days 7 to 14 and remains elevated through at least 8 weeks of age. Figure used with permission [11]