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. 2019 Dec 16;10:381. doi: 10.1186/s13287-019-1446-z

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — The schematic representation of mechanism by which MSC-derived exosomes containing miR-133b affect glioma cell activities. MSCs transfer miR-133b to glioma cells through exosomes to inhibit the EZH2 expression via suppression of the Wnt/β-catenin signaling pathway, whereby the glioma cell proliferation, migration, and invasion are diminished, and ultimately, the progression of glioma is attenuated