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. 2015 Jun 1;142(11):2002–2013. doi: 10.1242/dev.117358

Fig. 9.

Fig. 9.

Activation of Yki does not change the cell fate from eye to antenna. (A) The wild-type expression of Cut (Ct, green), a marker for antennal fate, is restricted to a circular domain in the antennal region, as well as in the cone cells of the eye field. (B) Misexpression of yki3SA using a bi-Gal4 driver (bi>yki3SA) at the DV margin suppresses the retinal differentiation, as evident from Elav (red) expression, but does not induce ectopic Cut expression. (C) Misexpression of yki3SA on the posterior margin of the eye disc using a dpp-Gal4 driver (dpp>yki3SA) results in suppression of Elav (red) as well as Ct (green) expression. (D-D‴) Random gain-of-function clones of yki3SA (hsFLP>yki3SA) marked by (D,D′) GFP (blue) reporter results in suppression (D,D″) Ct (green). (D,D‴) Elav expression (red) is downregulated in yki3SA clones. The orientation and magnification of imaginal discs and adult heads are identical in all panels.