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. 2019 Dec 18;10:5766. doi: 10.1038/s41467-019-13554-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Western blotting of the synaptosomal fraction from dorsal hippocampus of dnBMAL1 mice show significant reduction of phosphorylation level of GluA1 S845 (pS845). The graph represents fold changes compared to pS845 levels in WT. b, c Experimental designs are illustrated at the top of each panel. Memory retrieval is impaired at ZT10 in GluA1 S845A knockin (S845A) mice in b social recognition task (recognition index) and c contextual fear memory. dn dnBMAL1. All values are mean ± SEM. Individual data points are displayed as dots. *p < 0.05 as determined by one-way ANOVA with post hoc test. The results of the statistical analyses are presented in Supplementary Table 2. Source data are provided as a source data file.