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. 2019 Dec 19;12:76. doi: 10.1186/s13072-019-0318-1

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© The Author(s) 2019

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — RA caused upregulation of Hox genes. a RNA-seq analysis of the dynamic expression of the four Hox clusters in cranial neural tissue of RA-induced mouse NTDs embryos from E8.5 to E10.5. b Dynamic expression of the 10 Hox genes in cranial neural tissue of RA-induced mouse NTDs embryos from E8.5 to E10.5. c Hox gene mRNA in cranial neural tissue of RA-induced mouse NTDs embryos was measured by RT-qPCR. Actb was used as control. Data are shown as the mean (SD; n = 3). *P < 0.05