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. 2019 Dec 18;18:431. doi: 10.1186/s12936-019-3071-9

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© The Author(s) 2019

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Antiplasmodial activity of Terminalia albida treatment in ECM model. C57BL/6 mice were infected with P. berghei ANKA and treated 2 h later with chloroquine (5 mg/kg), Terminalia albida crude extract (100 mg/kg) or water (25 mL/kg) from Day 0 to Day 3. a Mean parasite densities during infection. b Percentages of parasite suppression according to the treatment, calculated by comparison to H₂O treated mice. **P < 0.005, ***P < 0.0005 compared Terminalia albida to H₂O group