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. 2019 Nov 14;19(1):7–16. doi: 10.3892/ol.2019.11093

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Molecular mechanisms underlying lncRNA H19 regulation in breast cancer. lncRNA, long non-coding RNA; SAHH, s-adenosylmethionine; SAH, s-adenosylhomocysteine; miR, microRNA; EGFR, epidermal growth factor receptor; Akt, protein kinase B; Erk, extracellular signal-regulated kinase; ceRNA, competing endogenous RNA; DNMT1, DNA methyltransferase 1; STAT3, signal transducer and activator of transcription 3; ICR, imprinting control region; CRD-BP, cMyc mRNA coding region instability determinant; CTCF, CCCTC-binding factor; IGF2, insulin-like growth factor 2.