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. 2019 Dec 21;9:204. doi: 10.1186/s13568-019-0924-0

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© The Author(s) 2019

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The effect of myricitrin on reactive oxygen species and malondialdehyde levels in high-cholesterol diet (HCD)-fed rats. The rats were divided into the following groups: sham (standard food), control (1% HCD), 1 μM myricitrin + 1% HCD, 10 μM myricitrin + 1% HCD, 100 μM myricitrin + 1% HCD, and the positive control (10 mg/kg body weight atorvastatin). Each group contained six rats. Myricitrin was given to the rats via oral gavage for 45 consecutive days. ^***P < 0.001 compared with the sham group; ^#P < 0.05, ^##P < 0.01, and ^###P < 0.001 compared with control rats