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. 2019 Dec 17;10:1630. doi: 10.3389/fpls.2019.01630

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Copyright © 2019 Wang, Lv, Han, Guan, Shi, Kang, Zhang, Cao, Li, Zhang, Wang and Zhang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The role of CPK33 in strigolactone-induced H₂O₂ production and H₂O₂-mediated stomatal closure in guard cells. (A) Effects of GR24 on H₂O₂ production in guard cells of WT plants, cpk33-1 and cpk33-2 mutants. Fluorescence intensities were normalized to those of controls that were taken as 100% for the indicated experiments. (B) Epidermal strips of WT plants with open stomata were exposed to the MES-KCl buffer containing different concentrations of H₂O₂. The stomatal apertures were measured and presented as means ± SE of three independent experiments. Bars with different letters represent statistically significant differences.