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. 2019 Nov 27;20(23):5968. doi: 10.3390/ijms20235968

Figure 2.

Figure 2

HSP27 is upregulated and phosphorylated in platelets during myocardial infarction. (A) HSP27 levels and phosphorylation (pHSP27) in four representative patients with nonischemic chest pain (controls, lanes 1–4) and four patients with myocardial infarction (MI) (lanes 5–8) are shown. β-actin served as loading control. (B) Quantitative analysis of HSP27 levels and phosphorylation (C) in the group of patients with myocardial infarction (MI) (n = 12) compared to controls (n = 12); * p < 0.05. Mean HSP27 and phospho-HSP27 to actin ratio for the control group was set as 1. The blot is representative of 12 independent patient pairs.