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. 2019 Dec 26;14(12):e0227097. doi: 10.1371/journal.pone.0227097

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© 2019 Imtiazul et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — (A) Neutrophil infiltration in the lungs was evaluated in ischemic AKI (S1 Fig). (B) Immunoblot of BCL-2-associated X protein (Bax), and B cell leukemia/lymphoma 2 (Bcl-2) were measured. Bax/Bcl-2 ratio in the lungs was identified in ischemic AKI. (C) Receptor for advanced glycation end products (RAGE), marker to reflect pneumocyte I injury in ischemic AKI. (D) Total protein changes in BAL in ischemic AKI. *p < 0.05 at each time point compared to control.