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. 2019 Dec 26;14(12):e0226714. doi: 10.1371/journal.pone.0226714

Fig 7. Graphical summary of phenotypes in Brca1-KPC, Palb2-KPC and Brca2-KPC pancreatic cancer mouse models.

Fig 7

Palb2- deficient pancreatic tumors exhibited Brca1- deficient tumor like- or Brca2- deficient tumor like- features in terms of the presence of different PDAC precursor lesions and the intermediate levels of ADM lesions and immune cells infiltrations, possibly due to its role as a linker between Brca1 and Brca2.