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. 2019 Nov 13;24(1):588–604. doi: 10.1111/jcmm.14766

Figure 1.

Figure 1

miR‐27a plays a regulatory role in PC via BTG2. A, Comparison results of the top 400 DEGs from http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE22780, http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE16515, http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE32676 and http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE91035. B and C, BTG2 expression in http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE22780 and http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE91035. D, BTG2 expression in PC from GEPIA database. E, The correlation between BTG2 expression and survival of PC patients, as analysed in GEPIA database. F, Comparisons of miRs that regulate BTG2 from miRWalk, TargetScan, mirDIP, DIANA and starBase. G, The expression of hsa‐miR‐27a in http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE28955. miR‐27a‐3p, microRNA‐27a‐3p; BTG2, B‐cell translocation gene 2; PC, pancreatic cancer; DEGs, differentially expressed genes