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. 2019 Dec 26;19:351. doi: 10.1186/s12935-019-1070-0

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© The Author(s) 2019

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — LINC00511 fostered the process of GC via targeting miR-625-5p/NFIX axis a RT-qPCR assays and western blot assays were conducted to detect the expression of NFIX in cells transfected with pcDNA3.1/NFIX. b, c The rescue effect of pcDNA3.1/NFIX on cell proliferation was tested in CCK8 and colony formation assay. d, e The rescue influence of pcDNA3.1/NFIX was evaluated on rate of apoptosis in flow cytometry analysis and TUNEL assays. f The rescue effect of pcDNA3.1/NFIX on protein in apoptosis was appraised in western blot. *P < 0.05, **P < 0.01