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. 2019 Dec 27;9:20017. doi: 10.1038/s41598-019-56436-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Histopathological sections and magnetic resonance microscopy images of an acute and a chronic MI experiment. Left panel showing acute MI images (A): Histopathological stain with haematoxylin-eosin (a,e), T1-weighted imaging (b,f), T2-weighted imaging (c,g) and T2*-weighted image (d,h) for remote and infarcted tissues respectively. Right panel showing chronic MI images (B): Histopathological stains with HE (i,n) and picrosirius red (j,o), T1-weighted imaging (k,p), T2-weighted imaging (l,q) and T2*-weighted imaging (m,r) for remote and infarcted tissues respectively. Connective tissue is marked with a black arrow, pericardium is marked with a white arrow and a vessel is marked with a black dot. Areas with and without necrosis (A) or fibrosis (B) (shown by histopathology) can be easily distinguished in terms of image intensity by magnetic resonance microscopy. Abbreviations: HE = haematoxylin-eosin; MI = myocardial infarction; T1 = longitudinal relaxation; T2 = transversal relaxation; T2* = effective transversal relaxation.