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. 2019 Dec 27;7:11. doi: 10.1186/s40170-019-0206-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Inhibition of BCR-ABL does impact on the glycolytic state of K562 cells, independent of HIF signaling and hypoxia. Glucose consumption (a) and lactate production (b) of K562 wt and ARNT knockout cells grown under normoxic and hypoxic growth conditions for 24 h cultured in the presence of increasing doses of imatinib