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. 2019 Dec 30;10:51. doi: 10.1186/s13229-019-0306-0

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Pedigree from which samples were derived for this study. GPD2 mutational status (GPD2m: indicates the presence of variant) and degree of ASD affectation are indicated. Black shading corresponds to the affected proband (AP) and his twin brother, dark grey to the intermediate phenotype sister (IS), light grey to the trait-affected brother (TB), and white to unaffected family members, including the unaffected mother (UM). * indicates that renal epithelial cells from these individuals were used to derive multiple clonal iPSC lines.