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. Author manuscript; available in PMC: 2021 Jan 1.

Published before final editing as: J Endocrinol. 2019 Jul 1:JOE-19-0182.R2. doi: 10.1530/JOE-19-0182

Figure 4. — (A) De novo motif analysis was performed on ChIP-seq data using HOMER. (B) Binding analysis of Glis3 ChIP-seq data indicated a preference for introns and promoters. Upstream = −10kb to −1kb relative to TSS, TSS proximal = −1kb to TSS, Gene body = TSS to TES, Intergenic = remaining sequences. (C,D) ChIP-seq binding on the Ins2 promoter, and the Slc2a2 promoter and enhancer. Peaks indicates the peaks called using HOMER.

Figure 4. — (A) De novo motif analysis was performed on ChIP-seq data using HOMER. (B) Binding analysis of Glis3 ChIP-seq data indicated a preference for introns and promoters. Upstream = −10kb to −1kb relative to TSS, TSS proximal = −1kb to TSS, Gene body = TSS to TES, Intergenic = remaining sequences. (C,D) ChIP-seq binding on the Ins2 promoter, and the Slc2a2 promoter and enhancer. Peaks indicates the peaks called using HOMER.