(
A) Representative images of immunohistochemistry experiments on lung sections from TKO mice 3 months and 6 months after SCLC initiation with Ad-CMV-Cre. None of the mice had metastases at the 3 month time point while all the mice analyzed had evidence of metastasis at the 6 month time point. The Tuj1 antibody marks neuronal tubulin and TAU is a marker of axons. Hematoxylin was used as a counterstain (purple). Scale bar, 100 μm. (
B) Quantification of (
A), with N = 30–32 tumors analyzed from N = 3 mice at the 3 month time point and N = 30 tumors analyzed from N = 3 mice at the 6 month time point. Percentages are indicated. (
C) Images of GAP43 immunohistochemistry experiments for representative metastases. Hematoxylin was used as a counterstain (purple). Out of 9 metastases analyzed, six showed positive staining for GAP43. Scale bar, 100 μm. (
D) Analysis of the expression of 12/13 genes in the selected list of genes involved in axonogenesis and neuronal migration comparing metastases to primary tumors in the Adeno-CMV-Cre TKO mouse model (from
Supplementary file 2–table 2 and from
Denny et al., 2016, and
Yang et al., 2018). The two groups (primary tumors ‘CMV Tum’ and metastases ‘CMV Met’) were first compared by two-way ANOVA and then each gene was compared by t-test.