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. Author manuscript; available in PMC: 2021 Feb 1.

Published in final edited form as: J Neurovirol. 2019 Jul 7;26(1):3–13. doi: 10.1007/s13365-019-00783-6

Figure 3. — A. CD4 recovery correlated positively with serum Aβ42/Aβ40 ratio in the HIV(+) group as a whole. This ratio represents the proportion between the main Aβ isoform constituent the amyloid plaques (Aβ-42) in brain, and the main AB isoform synthetized (Aβ40). In the HIV-1B group, but not on subtype C: there was moderate negative correlation between: B. current CD4 (cells/mm³) in blood and Aβ-42 (pg/mL) in serum; C. current CD4 (cells/mm³) in blood and Aβ-38 (pg/mL) in serum; D. current CD4 (cells/mm³) in blood and Aβ-40 (pg/mL) in serum; E. nadir CD4 (cells/mm³) in blood and Aβ-42 (pg/mL) in serum; F. nadir CD4 (cells/mm³) in blood and Aβ-Total (pg/mL) in serum.

Figure 3. — A. CD4 recovery correlated positively with serum Aβ42/Aβ40 ratio in the HIV(+) group as a whole. This ratio represents the proportion between the main Aβ isoform constituent the amyloid plaques (Aβ-42) in brain, and the main AB isoform synthetized (Aβ40). In the HIV-1B group, but not on subtype C: there was moderate negative correlation between: B. current CD4 (cells/mm³) in blood and Aβ-42 (pg/mL) in serum; C. current CD4 (cells/mm³) in blood and Aβ-38 (pg/mL) in serum; D. current CD4 (cells/mm³) in blood and Aβ-40 (pg/mL) in serum; E. nadir CD4 (cells/mm³) in blood and Aβ-42 (pg/mL) in serum; F. nadir CD4 (cells/mm³) in blood and Aβ-Total (pg/mL) in serum.