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. 2019 Nov 13;8(1):347–356. doi: 10.1080/21623945.2019.1690834

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© 2019 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — Fibrosis infiltration in peri-N from patients with CPA.

(a) Peri-N FN and COLI immunohistochemical and Masson’s staining (×400), NT peri-N, n = 5; CPA peri-N, n = 5. (b) Expression of genes associated with fibrosis in peri-N from CPA group (n = 8) and normotension group (n = 10). Scale bar, 50 µm. NT, normotension; CPA, cortisol-producing adenoma. peri-N, perirenal adipose tissue. (c) Expression of fibrosis genes in subcutaneous adipose tissue from CPA and normotension patients, n = 8. Sub-Q, subcutaneous adipose tissue, n = 10. *P < 0.05 vs normotension subjects.**P < 0.01 vs normotension subjects.