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. 2020 Jan 2;11(1):4. doi: 10.1038/s41419-019-2198-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a–e Western blot a and quantification b–e show that the increased expression of GRP78 b, CHOP c, cleaved-caspase-12 d, and RIP1 e induced by Arc knockdown after TNI were partially prevented by AIDA, but not by MPEP. f, g TUNEL staining f and quantification g show that the enhanced neuronal apoptosis induced by Arc knockdown after TNI was attenuated by AIDA, but not by MPEP. h, i PI staining h and quantification i show that the increased number of PI-positive cells induced by Arc knockdown after TNI was inhibited by AIDA, but not by MPEP. j Cell viability assay shows that the decreased cell viability induced by Arc knockdown after TNI was alleviated by AIDA, but not by MPEP. k LDH release assay shows that the increased LDH release induced by Arc knockdown after TNI was partially prevented by AIDA, but not by MPEP. Error bars indicate SEM (n = 6). *p < 0.05 vs. Control group. ^#p < 0.05 vs^. TNI + Si-Arc-3 group.