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. 2020 Jan 14;15:5. doi: 10.1186/s13020-019-0284-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Effect of LTF and CaD on NF-κB p65 and p38-MAPK pathways. a Western blot and quantitative measurement of retinal NF-κB(p65); Lane loading was normalized by reblotting with GAPDH. b Western blot and quantitative measurement of p38-MAPK and phosphorylated p38-MAPK. Lane loading was normalized by reblotting with GAPDH. The levels were expressed as p- p38-MAPK/p38-MAPK and normalized relative to control group (n = 4). (*p < 0.05: versus NC group; △p < 0.05: versus DM group)