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. 2020 Jan 9;10:1564. doi: 10.3389/fphys.2019.01564

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Copyright © 2020 Makkos, Szántai, Pálóczi, Pipis, Kiss, Poggi, Ferdinandy, Chatgilialoglu and Görbe.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Adult myocardial cell viability (A) was measured in normoxia or after simulated ischemia-reperfusion (SI/R) injury with/without hiChol1-3 supplements. Total ROS (B) and superoxide (C) level was measured too. Total ROS (B) and superoxide (C) level were also measured in normoxia or after SI/R. Data are expressed as mean ± SEM, in comparison to normoxia vehicle control group (100%). $p < 0.05 normoxia vehicle vs. SI/R vehicle (t-test); ^∗p < 0.05 vs. normoxia vehicle (one-way ANOVA, LSD post hoc); #p < 0.05 vs. SI/R vehicle (one-way ANOVA, LSD post hoc); n = 5–14 on (A,C); n = 3–5 on (B).