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. 2020 Jan 16;5:2. doi: 10.1038/s41525-019-0108-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Protein-coding genes with higher expression in proneural GSCs (showed in blue) or mesenchymal GSCs (showed in red) are exclusively associated with better versus poor prognosis in GBM patients with tumors of molecular subtypes proneural or mesenchymal subtypes of GBM, respectively. b Genes associated with survival in GBM patients are listed by type of alternative splicing event detected in the GSCs: ES, exon skipping; IR, intron retention; MXE, mutually exclusive exons; ASS, alternative splice sites 3′ or 5′. c Survival curves of four genes (presented in a) exclusively affecting patients with GBM proneural or mesenchymal GBM. Significant log-rank p-values and multivariate Cox p-values are shown in blue.