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. 2019 Dec 9;19(2):990–998. doi: 10.3892/etm.2019.8309

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Downregulation of MALAT1 attenuates heart damage in an AMI model rat. (A) The efficiency of MALAT1 knockdown was confirmed by reverse transcription-quantitative PCR. (B) The infarcted area of heart was evaluated by 2,3,5-triphenyl-2H-tetrazolium chloride staining. (C) The cardiac function of rats was evaluated. The data are represented as the mean ± standard deviation (n=10). **P<0.01, ***P<0.001. AMI, acute myocardial infarction; LVFS, left ventricular fractional shortening; LVID, left ventricular internal dimension; MALAT1, metastasis associated lung adenocarcinoma transcript 1; NC, negative control; siRNA, short interfering RNA.