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. 2019 Nov 22;11(12):1847. doi: 10.3390/cancers11121847

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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tRA and ATMi alleviated thickening and the inflammatory response of mice skin exposed to UV. (a) Representative bare skin (upper panel) and Masson-stained skin sections (lower panel) of C57BL/6 mice exposed to indicated treatments. (b) Analysis of epidermal thickness; each section was measured three times at different positions. Data are presented as means ± SEM. (c) Quantification of skin response to UV radiation with the LSR (local skin response) grading scale. (d) mRNA levels of IL-1b were determined via RT-PCR. (e) mRNA levels of IL-10 were determined via RT-PCR. (f) mRNA levels of TNF-α were determined via RT-PCR. (g) mRNA levels of INF-α were determined via RT-PCR. Data are presented as means ± SEM (n = 8, ** p < 0.01, *** p < 0.001).

tRA and ATMi alleviated thickening and the inflammatory response of mice skin exposed to UV. (a) Representative bare skin (upper panel) and Masson-stained skin sections (lower panel) of C57BL/6 mice exposed to indicated treatments. (b) Analysis of epidermal thickness; each section was measured three times at different positions. Data are presented as means ± SEM. (c) Quantification of skin response to UV radiation with the LSR (local skin response) grading scale. (d) mRNA levels of IL-1b were determined via RT-PCR. (e) mRNA levels of IL-10 were determined via RT-PCR. (f) mRNA levels of TNF-α were determined via RT-PCR. (g) mRNA levels of INF-α were determined via RT-PCR. Data are presented as means ± SEM (n = 8, ** p < 0.01, *** p < 0.001).