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. 2020 Jan 16;17:23. doi: 10.1186/s12974-019-1695-x

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© The Author(s). 2020

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Inhibition of NMDAR or calpain restored BDNF/TrkB signaling disruption on day 1 post-surgery. a Representative Western blots of SBDP in the hippocampus. β-tubulin was included as loading control. b Quantitative analysis of the ratio of the SBDP to αII-spectrin. c Representative Western blots of BDNF in the hippocampus. β-tubulin was included as loading control. d Quantitative analysis of BDNF levels. e Representative Western blots of TrkB-FL. β-tubulin was included as loading control. f Quantitative analysis of TrkB-FL levels. g Representative Western blots of TrkB-ICD. β-tubulin was included as loading control. h Quantitative analysis of TrkB-ICD levels. Data are presented as the mean ± SEM (n = 6). *p < 0.05 compared to the con + veh group (**p < 0.01, ***p < 0.001), ^#p < 0.05 compared to the sur + veh group (^##p < 0.01, ^###p < 0.001)