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. 2020 Jan 17;10:549. doi: 10.1038/s41598-019-57362-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of nifedipine on synaptic transmission. (A) Scatter plot of ISI vs. PSP amplitude under control conditions (normal ASW) (black circles) and in the presence of 10 μM nifedipine (green squares). Inset, the data shown in the main part of the figure plotted with a different scaling. Note that facilitation is observed under both conditions, but the facilitation observed in the presence of nifedipine is much less than the facilitation observed under control conditions. (B) Bar graphs plotting PSP amplitude at a short ISI (first bin of pooled data) vs. a long ISI (last bin) under control conditions (normal ASW) (black) and in the presence of 10 μM nifedipine (green). Note that the PSP_{first bin}/PSP_{last bin} ratio in nifedipine was much less than the PSP_{first bin}/PSP_{last bin} ratio under control conditions (i.e., 2.36 vs. 1.62). In all cases data are plotted as means ± SEMs.