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. 2020 Jan 21;11:398. doi: 10.1038/s41467-019-13928-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — a Histograms (mean + s.e.) showing carotid artery blood flow over the 40-min observation period following redox-induced arterial injury. vWF^RH/RH NSG male mice were either uninfused (PBS; n = 5) or infused with human platelets (hPLT) plus PBS (n = 4), tirofiban (Tiro.; n = 4), M-tirofiban (M-Tiro.; n = 4), or Hr10 (n = 4). b Blood loss (mean + s.e.) in vWF^RH/RHNSG male mice uninfused (PBS; n = 4) or infused with hPLT plus PBS (n = 4), tirofiban (Tiro.; n = 6), M-tirofiban (M-Tiro.; n = 4), or Hr10 (n = 4). p values are indicated. c Bleeding time analysis (mean +/− s.e.) in vWF^RH/RH NSG male mice treated as in Fig. 6b. Statistical tests by Student’s t-test, two-tailed; ns, not significant.