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. 2020 Jan 22;82(2):17. doi: 10.1007/s11538-019-00685-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 12 — Strain-induced PIN localisation does not significantly affect the formation of reverse flows when weighted below chemically induced localisation. Model (1)–(4b),(5) solved on a modified lattice of growing cells, with source and sink cells as in Fig. 11. a Strain-induced PIN localisation is weighted equally with flux-induced PIN localisation, $λ = ν = 0.25$ . Auxin flows from the source cells to halfway down the tissue where it branches out and then flows back up the outer cells. Auxin produced at the root tip also flows up the outer layer cells. b Strain-induced PIN localisation is weighted above flux-induced PIN localisation, $λ = 0.1$ , $ν = 0.4$ . Auxin flows directly from source cells to sink cells. Auxin produced in the central file of cells below the source cells does not flow to the base of the tissue, instead immediately flowing outwards to the outer cells where it then flows upwards to the sink cells. All parameters are described in Appendix Tables 2 and 3, with zero-flux boundary conditions. Cells are represented in the reference configuration (Color figure online)